FISIOPATOLOGIA DE LA DISPLASIA BRONCOPULMONAR PDF

Parte 1: Epidemiología, fisiopatología y clínica. Seguimiento neumológico de los niños con displasia broncopulmonar al alta de la Unidad de Cuidados. Epidemia de displasia broncopulmonar: incidencia y factores asociados en una cohorte de niños prematuros en Bogotá, Colombia. Juan G. Ruiz-Peláez1,2,3. Displasia Broncopulmonar. ES. eliana silva. Updated 6 September Transcript. Displasia Broncopulmonar Diagnostico general. Nesecidad de mantener.

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Es posible evitar las transfusiones de sangre en la anemia del prematuro. A Management guideline to reduce the frequency of blood transfusion in very- low-birth-weight infants.

Inflammation and bronchopulmonary dysplasia. The mechanisms involved in this abnormal alveolar development are not yet well understood 6 – 8.

Anti-Capase 3 is a rabbit polyclonal antibody, clone 3CSP03, 1: Stimulation of the Fas receptor results in its trimerization and recruitment of two key signaling proteins, the adapter protein Fas-associated death domain, FADD, and the initiator cysteine protease Caspase 8. In recent years, the frequency of this disease has risen mainly because of the increased survival of very low birth weight preterm neonates with disruption of vascular and lung development linked to functional alterations related to surfactant deficiency and immaturity.

There were no statistically significant differences in the degree of positivity for PCNA in alveolar cells between the groups, which were all positive for this marker as cell proliferation is normally present in the canalicular and saccular stages of lung development, corresponding to the 22 nd to 36 th weeks of gestation Pathogenesis and pathology of COPD.

Картинки: Displasia broncopulmonar fisiopatologia

A higher proliferation rate was observed in infants with CLD suggesting that longer exposure to mechanical ventilation may stimulates dipslasia proliferation.

The positive control HPF photomicrography was chosen fisiopahologia the “mask”, which contained adequate levels of positive tissue immunoexpression signal. Morphometric analysis The formalin-fixed paraffin-embedded lung tissue samples were reexamined and classified into one of the three groups according to the histopathological and morphometric changes without considering the clinical data: Delayed cord clamping in the interest of the newborn child.

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Inflammation and bronchopulmonary dysplasia: Cisplasia multiple roles of PTEN in tumor suppression. PCNA is a nuclear protein which acts as an accessory factor of deoxyribonucleic acid DNA polymerase delta, it is required for DNA replication and repair, and consequently for cell replication Apoptosis was involved in the pathophysiological of CLD. The formalin-fixed paraffin-embedded lung tissue samples were reexamined and classified into one of the three groups according to the histopathological brondopulmonar morphometric changes without considering the clinical data: The study was divided into three groups: The number of females is greater than males.

X 2 0,; gL 1; p 0, Death the Fas way: More or less the half of cases had a very early anemia and the independent factors associated with its appearance were the presence of a severe disease and to total volume of blood extracted. The samples were then incubated overnight with the following primary antibodies: TRADD was not submitted to morphometric analysis because most cases were negative for this protein.

For each of the four samples of each case a degree of positive staining was assigned as follows: Abstract Bronchopulmonary dysplasia chronic lung disease in infants constitutes a heterogeneous group of diseases with multifactorial etiology and pathogenesis. The clinical profile of the study population is shown in Tables 23 and 4.

The group “without” CLD was more premature at birth mean Ringer S, Sacher A. Anemia Neonatal dentro de las 24 horas de vida: These findings were confirmed by other authors in human, sheep, lamb and rabbit lungs 17 – 2028 – During lung development there is a natural equilibrium between apoptosis and cell proliferation Mechanical strain-induced proliferation and signaling in pulmonary epithelial H cells.

Apoptosis and cell proliferation are involved in the pathophisiology of CLD. To achieve the necessary control, and for a multidisciplinary follow-up, we will be addressing this disease with the objective of elaborating a performance plan when these newborns are at home after being discharged.

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Function and regulation of expression of pulmonary surfactant-associated proteins. June 29, ; Accepted: The presence of maternal pathology such as hypertensive disorders of pregnancy and gestational diabetes did not influence the incidence of CLD.

The pattern of staining was scored as follows: Fas-ligand-induced apoptosis of respiratory epithelial cells causes disruption of postcanalicular alveolar development.

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For each case, an average percentage of positive area was determined in 12 HPF images. Peng H, Tong XM. The observer did not have prior knowledge to which group the samples belong.

Apoptosis and cell proliferation are controlled physiological tissue repair processes regulated by fisiopatoloia and proteins. This is an Open Access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. However, the most important risk factor is the exposure to oxygen and mechanical ventilation 5.

Displasia Broncopulmonar by eliana silva on Prezi

Early vs delayed clamping of the umbilical cord in full term, preterm and very preterm infants. Its expression is increased in cells which are proliferating.

The severity of these disorders, however, has been modulated by changes in fisiopatlogia practice. Increased apoptosis “new” form and cell proliferation in lungs with CLD were observed in this study.

Premature infants with congenital abnormalities, chronic intrauterine diseases or meconium aspiration syndrome, as well as those whose medical records or samples were inadequate, were excluded. Apoptosis and proliferation in lungs of ventilated and oxygen-treated preterm infants. BPD is fisippatologia chronic lung disease, characterized by an impaired lung function due to a reduced final alveolar number and vascular growth.